(Last updated : 2019-04-18 16:08:55)
  Matsuoka Hidetada
   Department   School of Medicine  Physiology(2), Biomedical Sciences
   Position  
■ Journal
1. journal article  Differences among muscarinic agonists in M1 receptor-mediated nonselective cation channel activation and TASK1 channel inhibition in adrenal medullary cells. 2019/01 Link
2. journal article  Lack of p11 expression facilitates acidity-sensing function of TASK1 channels in mouse adrenal medullary cells. 2019/01 Link
3.   Expression and regulation of M-type K+ channel in PC12 cells and rat adrenal medullary cells. 2018/03 Link
4. journal article  Corticotropin-releasing hormone-binding protein is upregulated by brain-derived neurotrophic factor and is secreted in an activity-dependent manner in rat cerebral cortical neurons. 2018/01 Link
5. review  Muscarinic receptors in adrenal chromaffin cells: physiological role and regulation of ion channels. 2018/01 Link
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■ Presentation
1. Molecular mechanism for muscarinic receptor-mediated endocytosis of TASK1 channels in adrenal medullary cells  2018/03/28
2. Molecular mechanism for NGF-induced endocytosis of TASK1 channels in rat adrenal medullary cells and PC12 cells  2016/03/22
3. Src mediates endocytosis of TWIK-related acid-sensitive K+ channels in PC12 cells in response to nerve growth factor.  2014/11
4. Nerve growth factor-induced endocytosis of TWIK-related acid-sensitive K+1 channels in adrenal medullary cells and PC12 cells  2013/09
5. NGF-induced endocytosis of TASK1 channels in adrenal medullary cells and PC12 cells  2012/03
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■ Present specialized field
General physiology 
■ Academic background
1. 〔Doctoral course〕, Osaka University, Completed,
■ Business career
1. 2015/08~ School of Medicine Biomedical Sciences Physiology(2)
■ Department laboratory expense researcher number
90374991